Pediatrics in Review
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(Pediatrics in Review. 2007;28:77-78.)
© 2007 American Academy of Pediatrics

In Brief

Otitis Externa

The first 20% of the full text of this article appears below.

Evolution of Management Approaches for Otitis Externa. Dohar JE. Pediatr Infect Dis J. 2003;22 :299 –308[CrossRef][Medline] Otitis Externa Review. Beers SL, Abramo TJ. Pediatr Emerg Care. 2004;20 :250 –253[CrossRef][Medline] Clinical Practice Guideline: Acute Otitis Externa. Rosenfeld RM, Brown L, Cannon CR, et al. Otolaryngol Head Neck Surg. 2006;134 :S4 –S23[CrossRef][Medline]

Otitis externa (OE), more commonly known as swimmer’s ear, is predominantly a bacterial infection accompanied by inflammation and loss of skin integrity of the external ear canal. Although rare before 2 years of age, the peak prevalence occurs between 7 and 12 years. The most common infectious agents are Pseudomonas aeruginosa and Staphylococcus aureus, often coexisting, with yeast accounting for less than 1% of isolates. OE results most commonly from increased moisture in the ear canal due to high humidity, warm temperatures, and swimming. The moisture causes edema and skin breakdown, which allows bacterial proliferation. Predisposing factors include local trauma; swimming, especially in water that has high bacterial counts; irritation to the ear that may occur from trauma, a foreign body, or a hearing aid; severe dermatitis; or viral infections such as herpes simplex or varicella. The clinical presentation can range from mild inflammation to severe loss of skin integrity and maceration.

Patients usually present with acute onset of unilateral ear pain. Itching, a sense of fullness in the ear, . . . [Full Text of this Article]


Kimberly E. Stone, MD, MPH
 Johns Hopkins University School of Medicine
Baltimore, Md

Janet R. Serwint, MD, Consulting Editor, In Brief


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Clarification
Lawrence F Nazarian
Pediatrics in Review Online, 29 Mar 2007 [Full text]

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